A Near Drowning accident left this 2 year old girl in critical condition. After being intensively treated in the hospital, she was finally discharged 35 days following the accident, but still unresponsive to all stimuli. Fortunately, she was able to have access to hyperbaric oxygen therapy and began a series of 40 sessions over 38 days (a total of 78 days following the near drowning). Her recovery was immediate, and the MRI that was previously taken before being discharged from the hospital was nearly completely reversed following the short course of hyperbaric therapy. Most notably for hyperbaric physicians was the dosage of oxygen that was used to achieve these remarkable results. Each session consisted of 45 minutes at 1.3 ATA (mild hyperbaric therapy — mHBOT). Not only was the pressure a lower pressure protocol, but there was no other breathing apparatus used in the treatment, just regular air breathing. This case report is consistent with many others who have had ‘miraculous recovery’ following hyperbaric oxygen therapy. However, this particular case is an excellent one to give the hyperbaric community a wake up and clearly demonstrates the power of mild hyperbaric oxygen therapy. The powerful reversal of severe brain damage demonstrates the effectiveness of mHBOT, as seen in this case (New York Post release) "The brain is very sensitive to even slight changes in oxygen tension and this is one key reason why so many neurological conditions are positively affected by m-HBOT." Another key factor for this girl’s quick and nearly complete recovery is that she had quick access to hyperbaric oxygen therapy (within 3 months). Timing is everything and generally speaking, the quicker the access, the better the outcome, particularly if this procedure is applied within the first 2 years following brain trauma. Longer courses of hyperbaric therapy may need to be applied for injuries that have exceeded 2 years, as hyperbaric oxygen therapy is gaining widespread publicity for its neurological regenerative effects and stem cell release. The end result of "new brain tissue" now gives new hope for both recent and long-term brain injury survivors <view study> Read Less

A new study in the Journal of atherosclerosis and thrombosis published this year shows that low pressure hyperbaric oxygen therapy can be a very effective tool for helping those suffering from metabolic syndrome. Metabolic syndrome is a cluster of conditions — increased blood pressure, high blood sugar, excess body fat around the waist, and abnormal cholesterol or triglyceride levels — that occur together, increasing your risk of heart disease, stroke and diabetes. This study looked at only 1.25 ATA (low pressure hyperbarics) and found that this dosage was effective enough to produce a lowering of blood sugars, blood pressure, total cholesterol, and insulin. Many other positive benefits were noted <view study> Read Less

For many years, hyperbaric pressures of only 1.04 ATA were considered to be insignificant in the hyperbaric field. Moreover, pressures as high as 1.3 ATA were considered to have no extra therapeutic value and even deemed to be the "placebo dose" in many studies trying to determine the efficacy of hyperbaric oxygen therapy. We now have convincing evidence that this information is completely wrong! Over the past 2 decades, research has confirmed that these lower pressures actually can have large physiological benefits. From theory, to animal studies, and now to human studies; we can confirm that any pressures between 1.0 ATA and 1.3 ATA should all be considered hyperbaric therapy, delivering 'higher than normal' oxygen levels into the body. By obeying the gas-pressure laws of physics, any pressure above 1.0 ATA will allow extra oxygen to easily enter into the body. Therefore, any extra pressure is an extra dose of oxygen, and as the pressure increases, so does the extra dose of oxygen entering the body! Quite simply put, the purpose of hyperbaric oxygen therapy is to provide extra oxygen at levels above and beyond the body's natural means of getting this nutrient. Think of it as an "oxygen supplement." Every time you breathe inside an oxygen chamber, you are continuously taking another dose of the extra oxygen. This then is a real dose of extra oxygen, and therefore cannot be considered a placebo! In a recent publication, Paul Harch was able to very eloquently present these misconceptions in a detailed report rebutting a very poorly designed study published in the Journal of Neurotrauma. He was able to site numerous scientific documents validating the use of both low and very low pressures, clearly demonstrating that even pressures as low as 1.04 ATA had physiological effects. <view study>   Read Less

Major News -- A landmark study, in a mainstream and peer-reviewed hyperbaric medical journal has just been released, and it supports the use of lower pressure protocols for hyperbaric medicine. This is groundbreaking, because for over two decades now, there has been a constant and heated debate going on between hyperbaric physicians on the use of low pressure protocols, and whether or not they were as effective as higher pressures. Advocates of lower pressure protocols have clearly focused their opinion on the scientific rationale of the gas pressure laws of physics and say that ANY pressure will have a physiological effect - they also have growing support from empirical data. On the other hand, skeptics of lower pressure protocols say "where is the research", and they also quote that "most of the clinical research is at higher pressures". This study should finally put these to rest. Here, they looked at 3 different pressure protocols: 1.3 ATA (ambient air), 1.5 ATA (pure oxygen), and 1.75 ATA (pure oxygen). They then added all 3 pressure protocols to a standard intensive exercise program for CP children and followed them with for an 8-month period of time. The results were astonishing. Not only did they find that all 3 hyperbaric oxygen pressures gave significant improvements, but there was no difference between the pressures. More startling to skeptics of lower pressure protocols, the lower pressure protocol did not even use the conventional pure oxygen, and instead only used regular ambient air. Why this is groundbreaking is because typically in past studies, a lower pressure protocol using regular ambient air has been used as the placebo dose in studies. This is really the point of contention. The supporters of lower pressure protocols argue "how can you use 1.3 ATA ambient air as a placebo when the laws of physics clearly show that this is actually a treatment dosage" This study clearly supports their notion pressure is the key variable in hyperbaric oxygenation therapy. Pandoras box is open! <view study> Read Less

People still believe that hyperbaric oxygen therapy only works at higher pressures. Most of them are only practicing with higher pressure protocols and have not taken the time to go through the steady influx of all the recent data and research making its way into current medical journals and publications. If they did, they would very quickly be able to see that lower pressures can provide very powerful effects, and sometimes even more powerful than those observed with higher pressures; the latter was just the case in a landmark study released in 2013. This study was published in the UHMS journal (a mainstream, peer reviewed, hyperbaric medical journal) and looked at the difference between 2.4 ATA (higher pressure HBOT) and 1.5 ATA (lower pressure HBOT). The results were stunning in virtually all areas observed. Researchers observed 92 inflammatory genes and this is what they quoted:

"Interestingly, oxygen at 1.5 atm abs affected many genes much more strongly than oxygen at 2.4. The reasons for this effect are unknown, but it does raise questions about the most appropriate treatment pressures for inflammatory conditions"

It was landmark study for lower pressure protocols, because it clearly demonstrated that the lower pressure protocols (1.5 ATA) had much stronger effects in the inflammatory genes observed than the higher pressures (2.4 ATA). <view study>     Read Less